Search results for "Ventricular Remodeling"

showing 10 items of 61 documents

EpCAM and microvascular obstruction in patients with STEMI: a cardiac magnetic resonance study

2020

Abstract Introduction and objectives Microvascular obstruction (MVO) is negatively associated with cardiac structure and worse prognosis after ST-segment elevation myocardial infarction (STEMI). Epithelial cell adhesion molecule (EpCAM), involved in epithelium adhesion, is an understudied area in the MVO setting. We aimed to determine whether EpCAM is associated with the appearance of cardiac magnetic resonance (CMR)-derived MVO and long-term systolic function in reperfused STEMI. Methods We prospectively included 106 patients with a first STEMI treated with percutaneous coronary intervention, quantifying serum levels of EpCAM 24 hours postreperfusion. All patients underwent CMR imaging 1 w…

Malemedicine.medical_specialtyMagnetic Resonance Spectroscopymedicine.medical_treatmentMagnetic Resonance Imaging Cine030204 cardiovascular system & hematologyVentricular Function Left03 medical and health scienceschemistry.chemical_compoundPercutaneous Coronary Intervention0302 clinical medicineInternal medicinemedicineHumansIn patientcardiovascular diseasesMyocardial infarctionVentricular remodelingEnd-systolic volumeAgedEjection fractionbusiness.industryMicrocirculationPercutaneous coronary interventionStroke VolumeEpithelial cell adhesion moleculeGeneral MedicineMiddle AgedEpithelial Cell Adhesion Moleculemedicine.diseaseMagnetic Resonance ImagingchemistryCardiologyST Elevation Myocardial InfarctionFemalebusinessCardiac magnetic resonancehuman activitiesRevista Española de Cardiología (English Edition)
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ST2 and left ventricular remodeling after ST-segment elevation myocardial infarction: A cardiac magnetic resonance study.

2018

Background: The association of soluble interleukin-1 receptor-like 1 (ST2) with left ventricular (LV) remodeling is unclear in patients with a first ST-segment elevation myocardial infarction (STEMI). The objective of this work was to assess the relationship between ST2, a marker of inflammation, and cardiac magnetic resonance (CMR) imaging-derived LV remodeling after a first STEMI. Methods: We prospectively evaluated 109 patients with a first STEMI treated with primary percutaneous coronary intervention who had ST2 assessed 24 h post-reperfusion. All patients underwent CMR imaging 1 week and 6 months after STEMI. The independent associations between ST2, LV diastolic and systolic volume in…

AdultMalemedicine.medical_specialtyCardiac magnetic resonancemedicine.medical_treatmentDiastoleMagnetic Resonance Imaging Cine030204 cardiovascular system & hematologyCohort Studies03 medical and health sciencesPercutaneous Coronary Intervention0302 clinical medicineInternal medicinemedicineHumansST segmentProspective Studiescardiovascular diseases030212 general & internal medicineMyocardial infarctionVentricular remodelingEnd-systolic volumeAgedEjection fractionVentricular Remodelingbusiness.industryLeft ventricular remodelingPercutaneous coronary interventionMiddle AgedST2medicine.diseaseInterleukin-1 Receptor-Like 1 ProteinST-segment elevation myocardial infarctioncardiovascular systemCardiologyST Elevation Myocardial InfarctionFemaleCardiology and Cardiovascular MedicinebusinessCardiac magnetic resonanceBiomarkersFollow-Up Studies
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Treatment of Fabry's Disease With Migalastat: Outcome From a Prospective Observational Multicenter Study (FAMOUS).

2019

Fabry's disease (FD) is an X-linked lysosomal storage disorder caused by the deficient activity of the lysosomal enzyme alpha-galactosidase A (alpha-Gal A) leading to intracellular accumulation of globotriaosylceramide (Gb3). Patients with amenable mutations can be treated with migalastat, a recently approved oral pharmacologic chaperone to increase endogenous alpha-Gal A activity. We assessed safety along with cardiovascular, renal, and patient-reported outcomes and disease biomarkers in a prospective observational multicenter study after 12 months of migalastat treatment under real-world conditions. Fifty-nine (28 females) patients (34 (57.6%) pretreated with enzyme replacement therapy) w…

AdultMalemedicine.medical_specialty1-DeoxynojirimycinTime FactorsGlobotriaosylceramideRenal function030226 pharmacology & pharmacyGastroenterologyVentricular Function Left03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicineMigalastatGermanymedicineClinical endpointHumansPharmacology (medical)Genetic Predisposition to DiseaseProspective StudiesPharmacologySphingolipidsVentricular Remodelingbusiness.industryEnzyme replacement therapyMiddle Agedmedicine.diseaseFabry's diseaseFabry diseaseBlood pressureTreatment Outcomechemistry030220 oncology & carcinogenesisalpha-GalactosidaseMutationFabry DiseaseFemaleGlycolipidsbusinessBiomarkersGlomerular Filtration RateClinical pharmacology and therapeutics
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Role of cardiac dyssynchrony and resynchronization therapy in functional mitral regurgitation

2016

Functional mitral regurgitation (FMR) is a common complication of left ventricle (LV) dysfunction and remodelling. Recently, it has been recognized as an independent prognostic factor in both ischaemic and non-ischaemic LV dysfunctions. In this review article, we discuss the mechanisms through which cardiac dyssynchrony is involved in FMR pathophysiologic cascade and how cardiac resynchronization therapy (CRT) can have therapeutic effects on FMR by reverting specific dyssynchrony pathways. We analyse recent clinical trials focusing on CRT impact on FMR in 'real-world' patients, the limits and future perspectives that could eventually generate new predictors of CRT response in terms of FMR r…

medicine.medical_specialtyPacemaker Artificialmedicine.medical_treatmentCardiac resynchronization therapyVentricular dyssynchrony030204 cardiovascular system & hematologyCardiac resynchronization; Mitral regurgitation; Ventricular dyssynchrony; Cardiology and Cardiovascular Medicine; Radiology Nuclear Medicine and ImagingVentricular Function LeftCardiac Resynchronization Therapy03 medical and health sciencesVentricular Dysfunction Left0302 clinical medicineInternal medicineCardiac resynchronizationNuclear Medicine and ImagingmedicineHumansRadiology Nuclear Medicine and imaging030212 general & internal medicineVentricular remodelingVentricular dyssynchronyMitral regurgitationMitral regurgitationClinical Trials as TopicVentricular Remodelingbusiness.industryCardiac Pacing ArtificialMitral Valve InsufficiencyStroke VolumeGeneral MedicineStroke volumemedicine.diseasePrognosisReview articleEchocardiography Doppler ColorClinical trialmedicine.anatomical_structureTreatment OutcomeVentriclecardiovascular systemCardiologyRadiologybusinessCardiology and Cardiovascular MedicineRadiology
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Adipose Stromal Vascular Fraction Improves Cardiac Function in Chronic Myocardial Infarction Through Differentiation and Paracrine Activity

2012

Fresh adipose-derived cells have been shown to be effective in the treatment of acute myocardial infarction (MI), but their role in the chronic setting is unknown. We sought to determine the long-term effect of the adipose derived-stromal vascular fraction (SVF) cell transplantation in a rat model of chronic MI. MI was induced in 82 rats by permanent coronary artery ligation and 5 weeks later rats were allocated to receive an intramyocardial injection of 107 GFP-expressing fresh SVF cells or culture media as control. Heart function and tissue metabolism were determined by echocardiography and 18F-FDG-microPET, respectively, and histological studies were performed for up to 3 months after t…

Cardiac function curvemedicine.medical_specialtymedicine.medical_treatmentHeart VentriclesBiomedical EngineeringMyocardial Infarctionlcsh:MedicineAdipose tissue030204 cardiovascular system & hematologyRevascularizationRats Sprague-Dawley03 medical and health sciences0302 clinical medicineFibrosisInternal medicineParacrine CommunicationmedicineAdipocytesMyocardial RevascularizationAnimalsMyocardial infarctionAngiogenic ProteinsVentricular remodeling030304 developmental biology0303 health sciencesTransplantationTissue Inhibitor of Metalloproteinase-1Ventricular Remodelingbusiness.industrylcsh:RCell DifferentiationTissue Inhibitor of MetalloproteinasesCell BiologyStromal vascular fractionmedicine.diseaseRatsTransplantationDisease Models AnimalPhenotypeEchocardiographyPositron-Emission TomographyChronic DiseaseCardiologyCytokinesFemaleStromal CellsbusinessCell Transplantation
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Papillary muscle relocation and mitral annuloplasty in ischemic mitral valve regurgitation: midterm results.

2014

Objectives The surgical approach for ischemic mitral regurgitation remains unclear. Many studies are in favor of adding the subvalvular procedure to mitral annuloplasty to reduce recurrent mitral regurgitation. This study reports the clinical and echocardiographic outcomes of papillary muscle relocation combined with mitral annuloplasty.Methods From 2003, 115 patients with severe ischemic mitral regurgitation who underwent papillary muscle relocation plus nonrestrictive mitral annuloplasty and coronary artery bypass grafting were retrospective analyzed. Patients' mean age was 52 ± 12.8 years, New York Heart Association class III or IV was 71%, and preoperative left ventricular ejection frac…

Pulmonary and Respiratory MedicineAdultMalemedicine.medical_specialtyMitral Valve AnnuloplastyTime FactorsTime FactorMyocardial IschemiaPapillary MuscleSeverity of Illness IndexDisease-Free SurvivalVentricular Function LeftPostoperative ComplicationsRecurrenceRisk FactorsMitral valve annuloplastyInternal medicineMitral valveMedicineHumanscardiovascular diseasesVentricular remodelingPapillary muscleMitral regurgitationEjection fractionVentricular Remodelingbusiness.industryRisk FactorMedicine (all)Mitral Valve InsufficiencyMiddle AgedPapillary Musclesmedicine.diseaseSurgerymedicine.anatomical_structureTreatment OutcomeCardiologycardiovascular systemMitral ValveSurgeryFemalePostoperative ComplicationCardiology and Cardiovascular MedicinebusinessMitral valve regurgitationHumanArteryThe Journal of thoracic and cardiovascular surgery
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Bigger is not better: cortisol-induced cardiac growth and dysfunction in salmonids

2015

This is a Published Manuscript of an article published by Company of Biologists in Journal of Experimental Biology, available online: http://www.biologists.com/ Stress and elevated cortisol levels are associated with pathological heart growth and cardiovascular disease in humans and other mammals. We recently established a link between heritable variation in post-stress cortisol production and cardiac growth also in salmonid fish. A conserved stimulatory effect of the otherwise catabolic steroid hormone cortisol is likely implied, but has to date not been established experimentally. Furthermore, whereas cardiac growth is associated with failure of the mammalian heart, pathological cardiac h…

Male0301 basic medicinemedicine.medical_specialtyendocrine systemHydrocortisonePhysiologyGene ExpressionHeart failureCardiac performance030204 cardiovascular system & hematologyAquatic ScienceBiologyBioinformatics03 medical and health sciences0302 clinical medicineSDG 3 - Good Health and Well-beingInternal medicinemedicineAnimalsChronic stressCardiac OutputMolecular BiologySwimmingEcology Evolution Behavior and SystematicsVentricular RemodelingHeartStroke VolumeHypertrophyMyocardial hypertrophymedicine.diseasePeer reviewRainbow trout030104 developmental biologyEndocrinologyOncorhynchus mykissInsect ScienceHeart failureMyocardial hypertrophy/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingExperimental biologyChronic stressFemaleAnimal Science and ZoologyRainbow trout
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Overexpression of genes involved in lymphocyte activation and regulation are associated with reduced CRM-derived cardiac remodelling after STEMI

2021

Abstract Aims Lymphopenia after ST-segment elevation myocardial infarction (STEMI) correlates with deleterious cardiac consequences and worse prognosis. An in-depth examination of genes implicated in lymphocyte proliferation, activation and regulation and their association with short- and long-term cardiac structure and function is therefore of great interest. Methods Peripheral blood mononuclear cells were isolated from 10 control subjects and 64 patients with a first STEMI treated with primary percutaneous coronary intervention and submitted to cardiac magnetic resonance after 1 week and 6 months. mRNA expression of genes implicated in lymphocyte activation (CD25 and CD69) and regulation …

Antigens Differentiation T-LymphocyteMale0301 basic medicinemedicine.medical_specialtyLymphocytemedicine.medical_treatmentProgrammed Cell Death 1 ReceptorImmunologyGene Expressionchemical and pharmacologic phenomenaLymphocyte proliferationLymphocyte Activation03 medical and health sciences0302 clinical medicineAntigens CDInternal medicineHumansImmunology and AllergyMedicineCytotoxic T cellCTLA-4 AntigenLectins C-TypeIL-2 receptorMyocardial infarctionGeneAgedPharmacologyVentricular Remodelingbusiness.industryInterleukin-2 Receptor alpha SubunitPercutaneous coronary interventionHearthemic and immune systemsOdds ratioMiddle Agedmedicine.diseaseMagnetic Resonance ImagingPathophysiology030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisCancer researchLymphocyte activationLeukocytes MononuclearCardiologyST Elevation Myocardial InfarctionFemaleCardiology and Cardiovascular MedicinebusinessInternational Immunopharmacology
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Development and Long-Term Follow-Up of an Experimental Model of Myocardial Infarction in Rabbits

2020

Simple Summary Ischemic heart disease is one of the leading causes of death. A series of processes occur during acute myocardial infarction that contribute to the development of ventricular dysfunction, with subsequent heart failure and ventricular arrhythmias, which account for most episodes of sudden cardiac death in these patients. These complications are associated with the adverse cardiac remodeling that occurs during the healing process following an acute episode. The remodeling causes the appearance of a substrate that can trigger life-threatening arrhythmias, such as tachycardia and/or ventricular fibrillation. The development of experimental models for analyzing the basic mechanism…

Programmed stimulationmedicine.medical_specialtyLong term follow upmedicine.medical_treatment030204 cardiovascular system & hematologyischemia/reperfusion injuryventricular remodelingArticle03 medical and health sciencesexperimental surgery0302 clinical medicineRisk areaInternal medicinelcsh:ZoologymedicineMyocardial infarctionlcsh:QL1-991cardiovascular diseasesVentricular remodelingLigature030304 developmental biology0303 health scienceslcsh:Veterinary medicineGeneral Veterinarybusiness.industryExperimental modelventricular arrhythmiasInfarct sizemedicine.diseasemyocardial infarctionCardiologycardiovascular systemlcsh:SF600-1100Animal Science and Zoologycardiac mappingbusinessAnimals : an Open Access Journal from MDPI
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Recombinant growth hormone therapy in patients with ischemic cardiomyopathy : effects on hemodynamics, left ventricular function, and cardiopulmonary…

1999

Background —We studied the effects of recombinant growth hormone (rhGH) on exercise capacity and cardiac function in patients with ischemic cardiomyopathy. Methods and Results —Seven patients (aged 55±9 years) with mild to moderate congestive heart failure (ejection fraction 31±4%) who were on standard therapy were included. The patients were studied at baseline, after 3 months of rhGH treatment, and 3 months after rhGH discontinuation. Cardiac function was assessed by exercise capacity, right heart catheterization at rest and after submaximal exercise, MRI, echocardiography, and Holter monitoring. When administered at a dose of 2 IU/d, rhGH doubled the serum concentration of insulin-like …

Cardiac function curveMalemedicine.medical_specialtyCardiac outputCardiomyopathyMyocardial IschemiaHemodynamicsVentricular Function LeftPhysiology (medical)Internal medicinemedicineHumansVentricular remodelingIschemic cardiomyopathyEjection fractionExercise Tolerancebusiness.industryHuman Growth HormoneHemodynamicsMiddle Agedmedicine.diseaseCombined Modality TherapySurgeryHeart failureCardiologyCardiology and Cardiovascular MedicinebusinessCirculation
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